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Vol. 119, Issue 8, August 2010, pp.223-229

 

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Role of Catecholamine in Tumor Angiogenesis Linked to Capacitance Relaxation Phenomenon

 

1Guangyue SHI, 1Guanjie SUI, 2Jianxia JIANG, 3*T. K. BASAK

1Department of Medicine Oncology, The Third Affiliated Hospital of Harbin Medical University,

Harbin 150040, China

2Department of Gastroenterology, The First Affiliated Hospital of Nanjing Medical University.

Nanjing, 210029, China

3Krishna Engineering College, Ghaziabad, India

2E-mail: jjxcasey@yahoo.com.cn

 

 

Received: 28 April 2010   /Accepted: 17 August 2010   /Published: 31 August 2010

 

Abstract: The present paper deals with the CgA level during metastasis linked with Capacitance relaxation phenomenon in cancer cell. CgA co-stored and correlated by exocytosis with catecholamines is a precursor to peptides that exert feedback regulatory control on catecholamine secretion. It is to be noted that CgA was the most sensitive marker for detecting patients with tumor angiogenesis. The progressive rise in CgA increases with the tumor size and this fact has been correlated with the Capacitance relaxation phenomenon (T. K. Basak, US patent No. 5691178, 1997) in different stages. The experimental results of Capacitance relaxation phenomenon were given as inputs to a model for correlation with the CgA level. This model is a control system model, the output of which is the CgA level. It is to be noted that the model is simulated in MATLAB. The expression of tumorogenisis in prostate and liver is also linked to Capacitance relaxation phenomenon in respect of its correlation with the CgA level.

 

Keywords: CgA level, Capacitance relaxation phenomenon, Tumor angiogenesis

 

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